盐酸气体吸入致急性呼吸窘迫综合征1例报道

发布时间：2017-12-30 17:36

本文关键词：盐酸气体吸入致急性呼吸窘迫综合征1例报道　出处：《上海交通大学学报(医学版)》2015年12期 　论文类型：期刊论文

【摘要】：目的探讨盐酸气体吸入引起的急性呼吸窘迫综合征(ARDS)临床特点及治疗方法。方法报道1例双相水平气道正压通气(BIPAP)联合吡非尼酮(PFD)治疗盐酸气体吸入致ARDS患者的临床资料,复习文献探讨治疗特点。结果男性患者,33岁,因吸入浓盐酸气体后出现咳嗽、咳痰伴胸闷、气促就诊。CT示两肺弥漫性改变,手指脉搏血氧饱和度75%,以"吸入性肺炎,ARDS"收治入院,经BIPAP辅助通气、PFD抗纤维化等治疗后患者氧合改善,临床症状好转,两肺纤维灶减少。结论早期BIPAP的应用可以显著降低ARDS患者的插管率以及病死率。ARDS纤维化期予以PFD可以防止甚至逆转纤维化。
[Abstract]:Objective to investigate the hydrochloric acid gas inhalation induced acute respiratory distress syndrome (ARDS) clinical characteristics and treatment methods 1 cases reported bi level positive airway pressure ventilation (BIPAP) combined with pirfenidone (PFD) treatment of HCl gas inhalation clinical data of patients with ARDS induced by the literature review on the treatment of male patients. Results 33. Old, due to inhalation of hydrochloric acid gas after the cough, expectoration and chest tightness, shortness of breath, treatment.CT showed diffuse pulmonary changes two, 75% finger pulse oximetry, with aspiration pneumonia, ARDS admitted by BIPAP assisted ventilation, improve the oxygenation in patients with PFD anti fibrosis after treatment, clinical symptoms improved, two lung fiber lesions reduced. Conclusion early application of BIPAP can significantly reduce the intubation rate and mortality rate of patients with ARDS.ARDS PFD to fibrosis stage may prevent and even reverse fibrosis.

【作者单位】：上海交通大学医学院附属仁济医院南院呼吸科;上海交通大学医学院附属仁济医院呼吸科;
【分类号】：R563.8
【正文快照】： 急性呼吸窘迫综合征(acute respiratory distresssyndrome,ARDS)是以呼吸困难和进行性低氧血症为主要临床表现的急性起病的呼吸衰竭。病理生理改变以肺容量减少、肺顺应性下降、通气/血流比失调为主,晚期出现肺组织广泛纤维化。该病病死率高,近半数患者最终死于顽固的低氧血症

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