突触和抑郁症
发布时间：2018-10-12 19:26
【摘要】：正抑郁症,国际上习称为抑郁障碍,是一组慢性、高患病率、具有高致残率及致死率的精神疾病,有其相应的神经生物学基础和相关脑结构和功能的异常。基因易感性和环境(应激事件)是其发病机制的两个重要因素,基因与环境相互作用导致抑郁症,其生物学机制最终通路可能是神经可塑性[1]。神经可塑性是指大脑对各种内外刺激产生的与功能
[Abstract]:Positive depression, known internationally as depressive disorder, is a group of chronic, high prevalence, high disability rate and mortality of mental diseases, has its corresponding neurobiological basis and related brain structure and function abnormalities. Gene susceptibility and environment (stress events) are two important factors of its pathogenesis. The interaction between gene and environment leads to depression, and the biological mechanism may be neural plasticity [1]. Neural plasticity is the function of the brain in response to various internal and external stimuli
【作者单位】： 南昌大学第二附属医院神经内科;
【基金】：江西省青年科学基金资助项目(编号:20122BAB215035)
【分类号】：R749.4
[Abstract]:Positive depression, known internationally as depressive disorder, is a group of chronic, high prevalence, high disability rate and mortality of mental diseases, has its corresponding neurobiological basis and related brain structure and function abnormalities. Gene susceptibility and environment (stress events) are two important factors of its pathogenesis. The interaction between gene and environment leads to depression, and the biological mechanism may be neural plasticity [1]. Neural plasticity is the function of the brain in response to various internal and external stimuli
【作者单位】： 南昌大学第二附属医院神经内科;
【基金】：江西省青年科学基金资助项目(编号:20122BAB215035)
【分类号】：R749.4
【共引文献】
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