表面活性蛋白A缺乏加剧小鼠肾梗阻性损伤后的肾间质纤维化
发布时间：2019-07-28 17:25
【摘要】：正肾间质纤维化是几乎每种类型的慢性肾病不可避免的后果,然而其潜在机制尚未完全清楚。Tian等在既往的研究中确定,表面活性蛋白A(surfactant protein A,SP-A)是参与单侧输尿管梗阻(unilateral ureter obstruction,UUO)诱导的肾纤维化的一种新的蛋白因子。UUO诱导小鼠肾上皮中SP-A表达,可能是由于增加的酸性应激和炎症。有趣的是,SP-A缺乏加重UUO
[Abstract]:Positive renal interstitial fibrosis is an inevitable consequence of almost every type of chronic kidney disease, but its potential mechanism is not fully understood. Tian et al., in previous studies, determined that A (surfactant protein A, SP 鈮，
本文编号：2520220
[Abstract]:Positive renal interstitial fibrosis is an inevitable consequence of almost every type of chronic kidney disease, but its potential mechanism is not fully understood. Tian et al., in previous studies, determined that A (surfactant protein A, SP 鈮，
本文编号：2520220
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