外伤性脑积水的研究进展

发布时间：2018-06-08 08:00

本文选题：外伤性脑积水 + 发病机制　；参考：《河北医科大学》2017年硕士论文

【摘要】：创伤性颅脑损伤(Traumatic brain injury,TBI)是指因为外力作用于头部所致的颅脑损伤,包括头部打击、高处坠落、交通事故等。外伤性脑积水(post-traumatic hydrocephalus,PTH)是颅脑创伤后常见并发症之一。随着医学技术的发展,颅脑损伤患者的救治成功率的越来越高,PTH的发生率逐渐增高,PTH会严重地影响患者的生活质量及预后,对个人和社会造成严重的负担,因此医务工作者对PTH越来越重视。PTH的发病机制尚未完全明确,目前主流的观点认为脑脊液的分泌、吸收及循环通路的障碍是PTH发展的原因,但最近也有研究认为,脑脊液的渗透压负荷及脑脊液动力学的病理改变是脑积水发展的原因。明确PTH的发病机制可以使患者得到更加有效的预防及治疗。因此,更好的了解PTH的发生率、分类、发病机制、危险因素、诊断标准、预防、治疗及治疗后效果的评估、并发症的处理及PTH的治疗前景,对于PTH的治疗具有关键性作用。本文通过对大量的文献的归纳及总结将对上述内容逐一做综述。
[Abstract]:Traumatic craniocerebral injury (TBI) refers to craniocerebral injury caused by external force acting on the head, including head blow, falling from height, traffic accident and so on. Post-traumatic hydrocephalus PTH is one of the common complications of traumatic hydrocephalus. With the development of medical technology, the rate of successful treatment of patients with craniocerebral injury is higher and higher. PTH will seriously affect the quality of life and prognosis of patients, and will cause a serious burden on individuals and society. Therefore, medical workers pay more and more attention to PTH and the pathogenesis of PTH is not completely clear. At present, the mainstream view is that the obstacle of cerebrospinal fluid secretion, absorption and circulatory pathway is the reason for the development of PTH. The osmotic loading of cerebrospinal fluid and the pathological changes of cerebrospinal fluid dynamics are the reasons for the development of hydrocephalus. Understanding the pathogenesis of PTH can make patients more effective prevention and treatment. Therefore, a better understanding of PTH incidence, classification, pathogenesis, risk factors, diagnostic criteria, prevention, treatment and post-treatment evaluation, treatment of complications and PTH treatment prospects, PTH treatment plays a key role in the treatment of PTH. In this paper, a large number of literature will be summarized and summarized one by one.
【学位授予单位】：河北医科大学
【学位级别】：硕士
【学位授予年份】：2017
【分类号】：R651.15

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